Experts have previously suggested that SARS-CoV-2 (the COVID-19 causing virus) damages the heart muscle cells when it enters these cells through ACE2 receptors
The study explains that the angiotensin-converting enzyme 2 (ACE2) receptor binds with the SARS-CoV-2 virus through a receptor-binding domain.
As per the study, the expression of about eight genes such as the ones responsible for hypoxia, uncontrolled inflammation or cytokine storm, oxidative stress and vitamin K biosynthesis, increased with age, suggesting the higher susceptibility of the elderly to the SARS-CoV-2 virus
People with congenital heart disease may not be at high risk of developing severe symptoms of COVID-19, suggests study
The scientists from the Congenital Heart Disease Centre at Columbia University Vagelos College of Physicians and Surgeons conducted a review study on 7,000 individuals who were born with congenital heart disease
What makes coronavirus an 'ace' at invading cells? Blame its club-shaped protein spike and a molecule in the human body
In the months since the coronavirus stormed on to the world’s stage, millions have been infected and billions of other lives affected indirectly
Recent research suggests that men have a higher amount of ACE2 in their plasma, which is likely why they are more vulnerable to COVID-19.
ACE2 is found in the heart, kidneys and it is thought to play a role in how the infection progresses into the lungs.
The genome of COVID-19 is 86% similar to the SARS genome, yet the two strains have played out very differently.